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Retinal disorders v7.8 COQ8B Achchuthan Shanmugasundram Tag Q3_24_promote_green was removed from gene: COQ8B.
Tag Q3_24_NHS_review was removed from gene: COQ8B.
Retinal disorders v7.8 COQ8B Achchuthan Shanmugasundram commented on gene: COQ8B: The rating of this gene has been updated to green following NHS Genomic Medicine Service approval.
Retinal disorders v7.7 COQ8B Achchuthan Shanmugasundram Source NHS GMS was added to COQ8B.
Source Expert Review Green was added to COQ8B.
Rating Changed from Amber List (moderate evidence) to Green List (high evidence)
Retinal disorders v6.19 COQ8B Achchuthan Shanmugasundram Classified gene: COQ8B as Amber List (moderate evidence)
Retinal disorders v6.19 COQ8B Achchuthan Shanmugasundram Added comment: Comment on list classification: As reviewed by Siying Lin, there is sufficient evidence available for the association of this gene with green rating in the next GMS update.
Retinal disorders v6.19 COQ8B Achchuthan Shanmugasundram Gene: coq8b has been classified as Amber List (Moderate Evidence).
Retinal disorders v6.18 COQ8B Achchuthan Shanmugasundram changed review comment from: PMID:39226897 reported the identification of compound heterozygous variants in COQ8B gene in five individuals from four different families with non-syndromic retinitis pigmentosa. In total, five different variants were identified from these patients (p.Arg63Trp, p.Trp189Ter, p.Asp386Asn, p.Val442Met and p.Trp520Ter). In addition, cell-based analysis of recombinant proteins deriving from these genotypes showed in all cases a significant decrease in ligand-protein interaction compared to the wild type.; to: PMID:39226897 reported the identification of compound heterozygous variants in COQ8B gene in five individuals from four different families with non-syndromic retinitis pigmentosa. In total, five different variants were identified from these patients (p.Arg63Trp, p.Trp189Ter, p.Asp386Asn, p.Val442Met and p.Trp520Ter). In addition, cell-based analysis of recombinant proteins deriving from these genotypes showed in all cases a significant decrease in ligand-protein interaction compared to the wild type.

This gene has not yet been associated with retinal phenotype either in OMIM or in Gene2Phenotype.
Retinal disorders v6.18 COQ8B Achchuthan Shanmugasundram commented on gene: COQ8B: PMID:39226897 reported the identification of compound heterozygous variants in COQ8B gene in five individuals from four different families with non-syndromic retinitis pigmentosa. In total, five different variants were identified from these patients (p.Arg63Trp, p.Trp189Ter, p.Asp386Asn, p.Val442Met and p.Trp520Ter). In addition, cell-based analysis of recombinant proteins deriving from these genotypes showed in all cases a significant decrease in ligand-protein interaction compared to the wild type.
Retinal disorders v6.18 COQ8B Achchuthan Shanmugasundram Publications for gene: COQ8B were set to PMID 39226897
Retinal disorders v6.17 COQ8B Achchuthan Shanmugasundram Phenotypes for gene: COQ8B were changed from Retinal dystrophy to Retinitis pigmentosa, MONDO:0019200
Retinal disorders v6.16 COQ8B Achchuthan Shanmugasundram Tag Q3_24_promote_green tag was added to gene: COQ8B.
Tag Q3_24_NHS_review tag was added to gene: COQ8B.
Retinal disorders v6.16 COQ8B Achchuthan Shanmugasundram reviewed gene: COQ8B: Rating: GREEN; Mode of pathogenicity: None; Publications: 39226897; Phenotypes: Retinitis pigmentosa, MONDO:0019200; Mode of inheritance: BIALLELIC, autosomal or pseudoautosomal
Retinal disorders v6.5 COQ8B Siying Lin gene: COQ8B was added
gene: COQ8B was added to Retinal disorders. Sources: Literature
Mode of inheritance for gene: COQ8B was set to BIALLELIC, autosomal or pseudoautosomal
Publications for gene: COQ8B were set to PMID 39226897
Phenotypes for gene: COQ8B were set to Retinal dystrophy
Mode of pathogenicity for gene: COQ8B was set to Other
Review for gene: COQ8B was set to GREEN
Added comment: Recent publication of 4 families with non-syndromic retinal dystrophy associated with biallelic COQ8B variants, with cell-based analysis of recombinant proteins deriving from these genotypes, showing a significant decrease in ligand-protein interaction compared to the wild type.
Sources: Literature