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Hypertrophic cardiomyopathy v5.22 KLHL24 Arina Puzriakova reviewed gene: KLHL24: Rating: GREEN; Mode of pathogenicity: ; Publications: ; Phenotypes: ; Mode of inheritance:
Hypertrophic cardiomyopathy v5.18 KLHL24 Arina Puzriakova Tag Q3_24_promote_green was removed from gene: KLHL24.
Tag Q3_24_NHS_review was removed from gene: KLHL24.
Hypertrophic cardiomyopathy v5.18 KLHL24 Arina Puzriakova Source Expert Review Green was added to KLHL24.
Source NHS GMS was added to KLHL24.
Rating Changed from Amber List (moderate evidence) to Green List (high evidence)
Hypertrophic cardiomyopathy v4.16 KLHL24 Sarah Leigh Tag Q3_24_promote_green tag was added to gene: KLHL24.
Tag Q3_24_NHS_review tag was added to gene: KLHL24.
Hypertrophic cardiomyopathy v4.16 KLHL24 Sarah Leigh reviewed gene: KLHL24: Rating: GREEN; Mode of pathogenicity: None; Publications: ; Phenotypes: ; Mode of inheritance: None
Hypertrophic cardiomyopathy v4.16 KLHL24 Sarah Leigh Phenotypes for gene: KLHL24 were changed from Hypertrophic cardiomyopathy; Heart failure; arrhythmias; Risk of sudden death to Cardiomyopathy, familial hypertrophic, 29, with polyglucosan bodies, OMIM:620236; cardiomyopathy, familial hypertrophic, 29, with polyglucosan bodies, MONDO:0859372
Hypertrophic cardiomyopathy v4.15 KLHL24 Sarah Leigh Classified gene: KLHL24 as Amber List (moderate evidence)
Hypertrophic cardiomyopathy v4.15 KLHL24 Sarah Leigh Gene: klhl24 has been classified as Amber List (Moderate Evidence).
Hypertrophic cardiomyopathy v4.13 KLHL24 Nour Elkhateeb gene: KLHL24 was added
gene: KLHL24 was added to Hypertrophic cardiomyopathy. Sources: Literature
Mode of inheritance for gene: KLHL24 was set to BIALLELIC, autosomal or pseudoautosomal
Publications for gene: KLHL24 were set to 30715372; 32870709; 36672924
Phenotypes for gene: KLHL24 were set to Hypertrophic cardiomyopathy; Heart failure; arrhythmias; Risk of sudden death
Review for gene: KLHL24 was set to GREEN
Added comment: KLHL24 variants have been reported in relation to autosomal recessive hypertrophic cardiomyopathy in several individuals from four families in three publications (PMIDs: 30715372, 32870709, 36672924) with variants including missense and nonsense variants. The mechanism of pathogenicity is reported to be loss of function. This gene-disease relationship is also supported by expression data (PMID: 23715323).
Sources: Literature