Haematological malignancies cancer susceptibility

Gene: ADA

Amber List (moderate evidence)

ADA (adenosine deaminase)
EnsemblGeneIds (GRCh38): ENSG00000196839
EnsemblGeneIds (GRCh37): ENSG00000196839
OMIM: 608958, Gene2Phenotype
ADA is in 13 panels

2 reviews

Sarah Leigh (Genomics England Curator)

Green List (high evidence)

Numerous reports of ADA variants associated with Severe combined immunodeficiency due to ADA deficiency (OMIM: 102700), which has been associated with acute myeloid leukemia (AML)(PMID: 32098966).
Created: 7 May 2025, 2:27 p.m. | Last Modified: 7 May 2025, 2:27 p.m.
Panel Version: 4.8

Lauma Freimane (Children's Clinical University Hospital)

A causative factor of T cell-negative, B cell-negative, natural killer cell-negative severe combined immunodeficiency.
Sources: Literature
Created: 19 Jun 2023, 12:57 p.m.

Mode of inheritance
BIALLELIC, autosomal or pseudoautosomal

Phenotypes
severe combined immunodeficiency

Publications

History Filter Activity

7 May 2025, Gel status: 2

Set Phenotypes

Sarah Leigh (Genomics England Curator)

Phenotypes for gene: ADA were changed from severe combined immunodeficiency to Severe combined immunodeficiency due to ADA deficiency, OMIM: 102700

7 May 2025, Gel status: 2

Entity classified by Genomics England curator

Sarah Leigh (Genomics England Curator)

Gene: ada has been classified as Amber List (Moderate Evidence).

19 Jun 2023, Gel status: 0

Created, Added New Source, Set mode of inheritance, Set publications, Set Phenotypes

Lauma Freimane (Children's Clinical University Hospital)

gene: ADA was added gene: ADA was added to Haematological malignancies cancer susceptibility. Sources: Literature Mode of inheritance for gene: ADA was set to BIALLELIC, autosomal or pseudoautosomal Publications for gene: ADA were set to 32098966 Phenotypes for gene: ADA were set to severe combined immunodeficiency